JackOss <j...@o...pl> napisał(a):

> NewBe napisał(a):
> > Wie ktos moze, gdzie w sieci znajde wyniki sekcji zwlok autora i gury
> > prozdrowotnej diety niskotluszczowej Roberta Atkinsa?


> Powtazram za swoim "domowym" lekarzem, jednym z niewielu, o których
> wiem, że wiedzą b. dużo i nie zabierają głosu wiedzy NIE mając: Atkins
> zmarł na serce, a sekcji - bo sam zabronił - nie było.

Do informacji na temat smieci Atkinsa ktore podal Marcin, ja tylko dodam tyle,
ze kardiomiopatia na ktora on chorowal mogla byc zwiazana z niewlasciwa dieta
ktora on stosowal....

..kardiomiopatia jest bowiem zwiazana m.innymi z deficytem miedzi w
organizmie czlowieka. Deficyt ten moze byc wywolany nadmierna podaza cynku,
zelaza i wapnia w diecie (nadmiar produktow pochodzenia zwierzecego).

Deficyt cynku w polaczeniu z dieta wysokotluszczowa (bogata w tluszcze
nasycone) moze byc powodem pogorszenia nie tylko kardiomiopatii ale i innych
chorob serca.

Ponizej wklejam informacje na temat zwiazku kardiomiopatii z deficytem cynku
i nadmierna iloscia tluszczy w diecie:

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Nutr. 1996 Apr;126(4):807-16. Related Articles, Links

Aspects of cardiomyopathy are exacerbated by elevated dietary fat in
copper-restricted rats.

Jalili T, Medeiros DM, Wildman RE.

Department of Human Nutrition and Food Management, Ohio State University,
Columbus 43210, USA.

The objective of this study was to determine if a high fat diet having a
2:1 saturated-polyunsaturated fatty acid ratio exacerbates signs of copper
deficiency. Male weanling Long-Evans rats were randomly placed into one of the
following treatment groups: adequate copper low fat or deficient copper high
fat. The levels of fat used were 31 or 12% of daily energy, and copper
concentrations were 94.5 micromol/kg and <15.8 micromol/kg in the
copper-adequate and copper-deficient diets, respectively. Cardiac hypertrophy
as well as lower liver copper levels and superoxide dismutase activity were
observed in both groups of copper-deficient rats. Irrespective of copper
level, consumption of the high fat diet resulted in the thickening of the
interventricular septum and left ventricular free wall. Electrocardiograms
revealed that the copper-deficient high fat diet led to a significantly
smaller QT interval compared with all other groups. Significantly greater
S-wave voltage due to copper deficiency was observed. Significantly lower
heart cytochrome c oxidase (CCO) activity was found in the copper-deficient
groups with the copper deficient high fat group showing the lowest activity.
Western blots of the cardiac non-myofibrillar fraction demonstrated lower
amounts of CCO nuclear encoded peptides in the copper-deficient groups, with
the least amount seen in the copper-deficient high fat treatment. These data
suggest that a high level of dietary fat exacerbates some of the signs of
copper deficiency.
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Jeff


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